Up until now, researchers have believed that ED is generally caused by factors such as diabetes, cardiovascular disease, testosterone deficiency, anxiety, certain drugs, and so forth.
But for the first time, researchers have now tied it to a specific place in the human genome.
The study, published in the Proceedings of the National Academy of Sciences, showed that variations in a gene called SIM1 increased the risk of ED by a scary 26%.
The good news is that this can now be naturally and completely reversed.
This is how it works.
The SIM1 gene switches on when you get aroused and sends the required hormones to the right areas that trigger an erection.
If the SIM1 fails to switch on, then no erection happens.
Researchers examined the genetic information of 36,649 men from the Multiethnic Kaiser Permanente Northern California Genetic Epidemiology Research in Adult Health and Aging cohort and 222,358 men from the UK Biobank.
What they discovered was that for many men who suffered from erectile dysfunction, the mechanism that was used to turn on SIM1 did not work.
The interesting part is that the SIM1 is not connected to the body-mass index, heart diseases, low testosterone or any other secondhand factors that could lead to erectile dysfunction.
Its malfunction could therefore be attributed directly as a cause for erectile dysfunction.