We’ve known for years that saturated fat is one of the key causes of type 2 diabetes. We’ve also known that people suffering the disease have tan over-active immune response, loading their bodies with unneeded  inflammatory chemicals. But we haven’t known for sure how these two components interact to create type 2 diabetes.

Recent research from the University of North Carolina at Chapel Hill School of Medicine and published in the journal Nature Immunology sheds some light on this connection. Its findings show that only saturated fatty acids activate the immune system to produce special inflammatory protein named interleukin-1beta.

This inflammatory protein acts on several organism to turn off their response to insulin, which then leads to type 2 diabetes in the long haul.

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